A traumatic cardiac arrest is a cardiac arrest that occurs secondary to either blunt or penetrating trauma.  The most common cause of traumatic cardiac arrest is hemorrhage.  Other causes include tension pneumothorax, cardiac tamponade, and hypoxemia.  Although traumatic cardiac arrest has a high mortality rate, the neurological outcomes are better in those who survive compared to other causes of cardiac arrest.  Patients who have some signs of life upon the arrival of paramedics, or who initially present in pulseless electrical activity, and who subsequently achieve a return of spontaneous circulation, have the greatest probability of survival to hospital discharge.

Successful resuscitation requires simultaneous attention to assessment, airway management, and hemorrhage control. 

• Consider underlying medical causes of the cardiac arrest.
• Prioritize treatment of reversible causes over chest compressions in order of clinical precedence.
• Simultaneously attempt to identify and treat:
  • Hypovolemia
  • Hypoxemia
  • Tension pneumothorax
• Consider special circumstances.
• Consult with CliniCall to discuss treatment plan or early transport options.
• Consider discontinuing resuscitation efforts if interventions do not result in a return of spontaneous circulation.

• Interventions in traumatic cardiac arrests should be prioritized based on clinical relevance.  Paramedics should focus initially on controlling major hemorrhage through the appropriate use of direct pressure, tourniquets, and wound packing.
• Advanced airway management should not delay transport in urban areas where the traumatic arrest is the result of penetrating thoracic trauma, the presenting rhythm is PEA, and the time from loss of pulses to a trauma center is less than 15 minutes (20 minutes in the Vancouver Coastal-Urban region).
• Bilateral needle thoracentesis (or finger thoracostomy) should be performed on all traumatic arrests with blunt or penetrating chest trauma.  The preferred site for needle thoracentesis is the 5^th intercostal space in the mid-axillary line.  An alternative site is the 2^nd intercostal space on the mid-clavicular line, although this requires catheters longer than 5 cm.  Prehospital needle thoracentesis should be considered AGMP.  Although this is a low occurrence procedure, it does potentially expose the practitioner to an increased risk of exposure.  If this procedure is needed, crews are directed to proceed with airborne PPE including face-shield, EHFR/N95 mask, gown, and gloves.
• Obtain large-bore intravenous (or intraosseous) access and administer a bolus of 20 mL/kg.
• In blunt force cardiac arrest, a pelvic binder may be applied after addressing other reversible causes.  If a pelvic fracture is suspected of being a significant contributing factor, the binder should be placed earlier.

• The primary etiologies targeted by prehospital treatments include hypoxia, obstructive shock (specifically tension pneumothorax) and hypovolemia.
• Patients frequently present in an organized electrical rhythm on the monitor with no palpable pulses.  It has been shown that in these situations there is often a low perfusion state due to hypovolemia or vascular and cardiac obstruction preventing adequate perfusion. For management of major hemorrhage, volume replacement with large NS bolus or bilateral chest decompression may result in ROSC.
• Traumatic cardiac arrests with an initial rhythm of asystole, or wide complex PEA of less than 40 beats per minute are generally associated with poor outcomes.  It is reasonable to consider early discontinuation of resuscitation if there is no response to treatment.

Traumatic Arrest

Traumatic Arrest